[1] J. H. Barnett and J. W. Smoller, "The genetics of bipolar disorder," Neuroscience, vol. 164, pp. 331-343, 2009.
[2] Renk, Kimberly and White, "Bipolar disorder in children," Psychiatry journal, vol. 1, 2014.
[3] X. Wang, M. Xia and Y. Lai, "Disrupted resting-state functional connectivity in minimally treated chronic schizophrenia," Schizophrenia research, vol. 156, pp. 150-156, 2014.
[4] M. Li, T. Das, W. Deng, Q. Wang, Y. Li, L. Zhao, X. Ma and Y. Wang, "Clinical utility of a short resting-state MRI scan in differentiating bipolar from unipolar depression," Acta psychiatrica Scandinavica, vol. 136, pp. 288-299, 2017.
[5] V. Menon, "Large-scale brain networks and psychopathology: a unifying triple network model," Trends in cognitive sciences, vol. 15, pp. 483-506, 2011.
[6] L. Palaniyappan, G. Deshpande, P. Lanka and D. Rangaprakash, "Effective connectivity within a triple network brain system discriminates schizophrenia spectrum disorders from psychotic bipolar disorder at the single-subject level," Schizophrenia research, 2018.
[7] R. L. Buckner, J. R. Andrews-Hanna and D. L. Schacter, "The brain's default network: anatomy, function, and relevance to disease," Annals of the New York Academy of Sciences, vol. 1124, pp. 1-38, 2008.
[8] V. Menon and L. Q. Uddin, "Saliency, switching, attention and control: a network model of insula function," Brain Structure and Function, vol. 214, pp. 655-667, 2010.
[9] D. Sridharan, D. J. Levitin and V. Menon, "A critical role for the right fronto-insular cortex in switching between central-executive and default-mode networks," {Proceedings of the National Academy of Sciences, vol. 105, pp. 12569-12574, 2008.
[10]J. Long, Q. Xie, Q. Ma, M. Urbin and L. Liu, "Distinct interactions between fronto-parietal and default mode networks in impaired consciousness," Scientific reports, vol. 6, p. 38866, 2016.
[11]M.-J. Liang, Q. Zhou, K.-R. Yang and X.-L. Yang, "Identify changes of brain regional homogeneity in bipolar disorder and unipolar depression using resting-state FMRI," PloS one, vol. 8, p. e79999, 2013.
[12]C.-H. Liu, F. Li, S.-F. Li and Y.-J. Wang, "Abnormal baseline brain activity in bipolar depression: a resting state functional magnetic resonance imaging study," Psychiatry Research: Neuroimaging, vol. 203, pp. 175-179, 2012.
[13]C. Vargas, C. Lopez-Jaramillo and E. Vieta, "A systematic literature review of resting state network—functional MRI in bipolar disorder," Journal of affective disorders, vol. 150, pp. 727-735, 2013.
[14]K. Xu, H. Liu, H. Li and Y. Tang, "Amplitude of low-frequency fluctuations in bipolar disorder: a resting state fMRI study," Journal of affective disorders, vol. 152, pp. 237-242, 2014.
[15]D. Ongur, M. Lundy, I. Greenhouse and A. K. Shinn, "Default mode network abnormalities in bipolar disorder and schizophrenia," Psychiatry Research: Neuroimaging, vol. 183, pp. 59-68, 2010.
[16]R. Goya-Maldonado, K. Brodmann, M. Keil and S. Trost, "Differentiating unipolar and bipolar depression by alterations in large-scale brain networks," Human brain mapping, vol. 37, pp. 808-818, 2016.
[17]P. Magioncalda, M. Martino, B. Conio and A. Escelsior, "Functional connectivity and neuronal variability of resting state activity in bipolar disorder—reduction and decoupling in anterior cortical midline structures," Human brain mapping, vol. 36, pp. 666-682, 2015.
[18]N.-F. Jie, M.-H. Zhu, X.-Y. Ma and E. A. Osuch, "Discriminating bipolar disorder from major depression based on SVM-FoBa: efficient feature selection with multimodal brain imaging data," IEEE transactions on autonomous mental development, vol. 7, pp. 320-331, 2015.
[19]B. Rashid, M. R. Arbabshirani, E. Damaraju and M. S. Cetin, "Classification of schizophrenia and bipolar patients using static and dynamic resting-state fMRI brain connectivity," Neuroimage, vol. 134, pp. 645-657, 2016.
[20]S. Teng, C.-F. Lu, P.-S. Wang and C.-I. Hung, "Classification of bipolar disorder using basal-ganglia-related functional connectivity in the resting state," in 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC), IEEE, 2013, pp. 1057-1060.
[21]M. Li, T. a. D. W. Das and Q. Wang, "Clinical utility of a short resting-state MRI scan in differentiating bipolar from unipolar depression," Acta psychiatrica Scandinavica, vol. 136, pp. 288-299, 2017.
[22]Anticevic, M. W. Cole, G. Repovs and J. D. Murray, "Characterizing thalamo-cortical disturbances in schizophrenia and bipolar illness," Cerebral cortex, vol. 24, pp. 3116-3130, 2013.
[23]Y. Du, G. D. Pearlson, J. Liu and J. Sui, "A group ICA based framework for evaluating resting fMRI markers when disease categories are unclear: application to schizophrenia, bipolar, and schizoaffective disorders," Neuroimage, vol. 122, pp. 272-280, 2015.
[24]J. Gong, G. Chen, Y. Jia and S. Zhong, "Disrupted functional connectivity within the default mode network and salience network in unmedicated bipolar II disorder," Progress in Neuro-Psychopharmacology and Biological Psychiatry, vol. 88, pp. 11-18, 2019.
[25]R. A. Poldrack, E. Congdon, W. Triplett and K. Gorgolewski, "A phenome-wide examination of neural and cognitive function," Scientific data, vol. 3, p. 160110, 2016.
[26]G. S. Alexopoulos, M. J. Hoptman, D. Kanellopoulos and C. F. Murphy, "Functional connectivity in the cognitive control network and the default mode network in late-life depression," Journal of affective disorders, vol. 139, pp. 56-65, 2012.
[27]R. L. Bluhm, C. R. Clark, A. C. McFarlane and K. A. Moores, "Default network connectivity during a working memory task," Human brain mapping, vol. 32, pp. 1029-1035, 2011.
[28]R. Jacobs, A. Barba, J. Gowins and H. Klumpp, "Decoupling of the amygdala to other salience network regions in adolescent-onset recurrent major depressive disorder," Psychological medicine, vol. 46, pp. 1055-1067, 2016.
[29]C. Kelly, A. Di Martino, L. Q. Uddin and Z. Shehzad, "Development of anterior cingulate functional connectivity from late childhood to early adulthood," Cerebral cortex, vol. 19, pp. 640-657, 2008.
[30]D. S. Margulies, A. C. Kelly, L. Q. Uddin and B. B. Biswal, "Mapping the functional connectivity of anterior cingulate cortex," Neuroimage, vol. 37, pp. 579-588, 2007.
[31]C. McCabe and Z. Mishor, "Antidepressant medications reduce subcortical--cortical resting-state functional connectivity in healthy volunteers," Neuroimage, vol. 57, pp. 1317-1323, 2011.
[32]J. N. Pannekoek, I. M. Veer, M.-J. van Tol and S. J. van der Werff, "Aberrant limbic and salience network resting-state functional connectivity in panic disorder without comorbidity," Journal of affective disorders, vol. 145, pp. 29-35, 2013.
[33]H. McCarthy, N. Skokauskas, A. Mulligan and G. Donohoe, "Attention network hypoconnectivity with default and affective network hyperconnectivity in adults diagnosed with attention-deficit/hyperactivity disorder in childhood," JAMA psychiatry, vol. 70, pp. 1329-1337, 2013.
[34]Y. I. Sheline, J. L. Price, Z. Yan and M. A. Mintun, "Resting-state functional MRI in depression unmasks increased connectivity between networks via the dorsal nexus," Proceedings of the National Academy of Sciences, vol. 107, pp. 11020-11025, 2010.
[35]M. Vinod, "Large-Scale brain networks and psycopathology: a unifying triple network model," Trends in cognitive sciences, vol. 15, pp. 483-506, 2011.
[36]M. Vinod, "The Triple Network Model, Insight, and Large-Scale Brain Organization in Autism," Biological psychiatry, vol. 10, pp. 61-74, 1999.
[37]J. Platt and others, "Probabilistic outputs for support vector machines and comparisons to regularized likelihood methods," Advances in large margin classifiers, vol. 84, pp. 236, 2018.
[38]"The angular gyrus: multiple functions and multiple subdivisions," Seghier, Mohamed L, vol. 19, pp. 43-61, Seghier, Mohamed L.
[39]D. Lv, W. Lin, Z. Xue and W. Pu, "Decreased functional connectivity in the language regions in bipolar patients during depressive episodes but not remission," Journal of affective disorders, vol. 197, pp. 116-124, 2016.
[40]D. Lv, W. Lin, Z. Xue and W. Pu, "Decreased functional connectivity in the language regions in bipolar patients during depressive episodes but not remission," Journal of affective disorders, vol. 197, pp. 116-124, 2016.
[41]Y. Wang, S. Zhong, Y. Jia and Y. Sun, "Disrupted resting-state Functional connectivity innonmedicated Bipolar Disorder," Radiology, vol. 280, pp. 529-536, 2016.
[42]Hahn, P. Stein, C. Windischberger and A. Weissenbacher, "Reduced resting-state functional connectivity between amygdala and orbitofrontal cortex in social anxiety disorder," Neuroimage, vol. 56, pp. 881-889, 2011.
[43]F. Wang, J. H. Kalmar, Y. He and M. Jackowski, "Functional and structural connectivity between the perigenual anterior cingulate and amygdala in bipolar disorder," Biological psychiatry, vol. 66, pp. 516-521, 2009.
[44]D. G. Amaral and J. Price, "Amygdalo-cortical projections in the monkey (Macaca fascicularis)," Journal of Comparative Neurology, vol. 230, pp. 465-496, 1984.
[45]D. Pandya, G. Van Hoesen and M.-M. Mesulam, "Efferent connections of the cingulate gyrus in the rhesus monkey," Experimental brain research, vol. 42, pp. 319-330, 1981.
[46]Etkin, T. Egner, D. M. Peraza and E. R. Kandel, "Resolving emotional conflict: a role for the rostral anterior cingulate cortex in modulating activity in the amygdala," Neuron, vol. 51, pp. 871-882, 2006.
[47]J. N. Bae, J. R. MacFall, K. R. R. Krishnan and M. E. Payne, "Dorsolateral prefrontal cortex and anterior cingulate cortex white matter alterations in late-life depression," Biological psychiatry, vol. 60, pp. 1356-1363, 2006.
[48]H. He, Q. Yu, Y. Du and V. Vergara, "Resting-state functional network connectivity in prefrontal regions differs between unmedicated patients with bipolar and major depressive disorders," Journal of affective disorders, vol. 190, pp. 483-493, 2016.
[49]T. Onitsuka, M. E. Shenton, D. F. Salisbury and C. C. Dickey, "Middle and inferior temporal gyrus gray matter volume abnormalities in chronic schizophrenia: an MRI study," American Journal of Psychiatry, vol. 161, pp. 1603-1611, 2004.
[50]P. Magioncalda, M. Martino, B. Conio and A. Escelsior, "Functional connectivity and neuronal variability of resting state activity in bipolar disorder—reduction and decoupling in anterior cortical midline structures," Human brain mapping, vol. 36, pp. 666-682, 2015.
[51]Q. Zhou, F. Y. Womer, L. Kong and F. Wu, "Trait-Related Cortical-Subcortical Dissociation in Bipolar Disorder: Analysis of Network Degree Centrality.," The Journal of clinical psychiatry, vol. 78, pp. 584-591, 2017.